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ironjustice · Guest

On Nov 28, 6:50 am, anonym...@nowhere.you.know wrote: snip <<

You were told to stay off my threads dioxin boy ..

Who loves ya.
Tom

Jesus Was A Vegetarian!
http://tinyurl.com/2r2nkh

Man Is A Herbivore!
http://tinyurl.com/a3cc3

DEAD PEOPLE WALKING
http://tinyurl.com/zk9fk

ironjustice · Guest

On Nov 27, 5:26 pm, m...@otr.com wrote:
"Substantiates 7 nicotinic acetylcholine receptor in schizophrenia"
Not according to what the abstract you posted said:
"Recent studies indicate an association of the neuronal 7 nicotinic
acetylcholine receptor (7 AChR) with several aspects of
schizophrenia,
and decreased levels of this receptor in postmortem brains of
schizophrenic patients have been reported. In view of that and in"

More not less receptors should be associated with increased smoking.

I should read your post more .. closely ..

The should part ..

As pointed out .. they smoke four times more so there has to be a
connection to the nicotine receptors .. ?

Your comment I should have weighed with the known evidence of ..
**extreme** need for a smoke .. ?

Sooo .. would a decrease of receptors lead one to smoke an **extreme**
amount BECAUSE the receptors are not there .. ? .. IE: inhibition of
a7 nAChR

Which again would give credence to the lack of receptors DUE TO the
accumulated iron.

You smoke BECAUSE of a lack of receptors in order to simply function.

"In addition, inhibition of a7 nAChR at protein level was observed
in the cells exposed to high amounts of iron."

Who loves ya.
Tom

Jesus Was A Vegetarian!
http://tinyurl.com/634q5a

Man Is A Herbivore!
http://tinyurl.com/4rq595

DEAD PEOPLE WALKING
http://tinyurl.com/zk9fk

ironjustice@aol.com · Guest

On Nov 29, 5:58 pm, ironjustice <teamtan...@hotmail.com> wrote:More
not less receptors should be associated with increased smoking. <<

It seems this smoking IS .. coincidentally common ALSO to .. ADHD and
Parkinsons' .

"More than half of Parkinson>s patients smoke, as do half of
adolescents with severe Attention Deficit Hyperactivity Disorder
(ADHD)."

Therefore associated and evidenced to be affected when one targets the
acetylcholine pathway / IE: nicotine and choline activate
receptors ...

IF the 61 year old woman with "brain iron accumulation" .. was ..
ACTUALLY .. simply Parkinsons' / misdiagnosis / common cause .. "

One can OBVIOUSLY **see** .. her cure JUST like in MS and marijuana /
IE: tremors and symptoms disappear .

No denying what you .. see.

Iron COULD 'possibly' .. be involved in diseases which may .. share ..
a common treatment and therefore possibly a common .. cause.

Parkinsons and / or brain iron accumulation is reversed by iron
removal and they walk and talk normally PROVING .. imho .. a cure can
be effected.

THEREFORE a cure should be and can be .. expected .. IN .. OTHER ..
diseases which may share a common .. cause .. IE: iron ..

Parkinsons .. ? .. or brain iron accumulation .. ? .. or
schizophrenia .. ? or ADHD .. ? .. or Alzheimers .. ?

http://www.cosmosmagazine.com/features/print/2349/nicotine-can-it-sav...

Nicotine: can it save your brain?
Issue 23 of Cosmos, October 2008
by Becky McCall

-----------------

Who loves ya.
Tom

Jesus Was A Vegetarian!
http://tinyurl.com/2r2nkh

Man Is A Herbivore!
http://tinyurl.com/a3cc3

DEAD PEOPLE WALKING
http://tinyurl.com/zk9fk

[quote]On Nov 27, 5:26 pm, m...@otr.com wrote:
"Substantiates 7 nicotinic acetylcholine receptor in schizophrenia"
Not according to what the abstract you posted said:
"Recent studies indicate an association of the neuronal 7 nicotinic
acetylcholine receptor (7 AChR) with several aspects of
schizophrenia,
and decreased levels of this receptor in postmortem brains of
schizophrenic patients have been reported. In view of that and in"

More not less receptors should be associated with increased smoking.

I should read your post more .. closely ..

The should part ..

As pointed out .. they smoke four times more so there has to be a
connection to the nicotine receptors .. ?

Your comment I should have weighed with the known evidence of ..
**extreme** need for a smoke .. ?

Sooo .. would a decrease of receptors lead one to smoke an **extreme**
amount BECAUSE the receptors are not there .. ? .. IE: inhibition of
a7 nAChR

Which again would give credence to the lack of receptors DUE TO the
accumulated iron.

You smoke BECAUSE of a lack of receptors in order to simply function.

"In addition, inhibition of a7 nAChR at protein level was observed
in the cells exposed to high amounts of iron."

Who loves ya.
Tom

Jesus Was A Vegetarian!http://tinyurl.com/634q5a

Man Is A Herbivore!http://tinyurl.com/4rq595

DEAD PEOPLE WALKINGhttp://tinyurl.com/zk9fk[/quote]

ironjustice@aol.com · Guest

On Nov 30, 6:09 am, "ironjust...@aol.com" <ironjust...@aol.com> wrote:
"More than half of Parkinson>s patients smoke, as do half of
adolescents with severe Attention Deficit Hyperactivity Disorder
(ADHD)." <<

http://www.thelecithinstore.com/shop/custom.asp?recid=7

Brain biochemists are finding increasing evidence that a variety of
brain and bodily disorders may result from the inability of the brain
to produce or utilize sufficient acetylcholine.

The impact of these discoveries was summarized by one of the MIT
researchers when he recommended at a symposium on Dietary choline
Sources and Brain Function held at MIT in November 1977, “it comes as
a surprise that nature would allow our dietary habits to control such
a vital function as neurotransmitter synthesis.”
-----------------

Who loves ya.
Tom

Jesus Was A Vegetarian!
http://tinyurl.com/2r2nkh

Man Is A Herbivore!
http://tinyurl.com/a3cc3

DEAD PEOPLE WALKING
http://tinyurl.com/zk9fk

[quote]On Nov 29, 5:58 pm, ironjustice <teamtan...@hotmail.com> wrote:More
not less receptors should be associated with increased smoking.

It seems this smoking IS .. coincidentally common ALSO to .. ADHD and
Parkinsons' .

"More than half of Parkinson>s patients smoke, as do half of
adolescents with severe Attention Deficit Hyperactivity Disorder
(ADHD)."

Therefore associated and evidenced to be affected when one targets the
acetylcholine pathway / IE: nicotine and choline activate
receptors ...

IF the 61 year old woman with "brain iron accumulation" .. was ..
ACTUALLY .. simply Parkinsons' / misdiagnosis / common cause .. "

One can OBVIOUSLY **see** .. her cure JUST like in MS and marijuana /
IE: tremors and symptoms disappear .

No denying what you .. see.

Iron COULD 'possibly' .. be involved in diseases which may .. share ..
a common treatment and therefore possibly a common .. cause.

Parkinsons and / or brain iron accumulation is reversed by iron
removal and they walk and talk normally PROVING .. imho .. a cure can
be effected.

THEREFORE a cure should be and can be .. expected .. IN .. OTHER ..
diseases which may share a common .. cause .. IE: iron ..

Parkinsons .. ? .. or brain iron accumulation .. ? .. or
schizophrenia .. ? or ADHD .. ? .. or Alzheimers .. ?

http://www.cosmosmagazine.com/features/print/2349/nicotine-can-it-sav...

Nicotine: can it save your brain?
Issue 23 of Cosmos, October 2008
by Becky McCall

-----------------

Who loves ya.
Tom

Jesus Was A Vegetarian!http://tinyurl.com/2r2nkh

Man Is A Herbivore!http://tinyurl.com/a3cc3

DEAD PEOPLE WALKINGhttp://tinyurl.com/zk9fk

On Nov 27, 5:26 pm, m...@otr.com wrote:
"Substantiates 7 nicotinic acetylcholine receptor in schizophrenia"
Not according to what the abstract you posted said:
"Recent studies indicate an association of the neuronal 7 nicotinic
acetylcholine receptor (7 AChR) with several aspects of
schizophrenia,
and decreased levels of this receptor in postmortem brains of
schizophrenic patients have been reported. In view of that and in"

More not less receptors should be associated with increased smoking.

I should read your post more .. closely ..

The should part ..

As pointed out .. they smoke four times more so there has to be a
connection to the nicotine receptors .. ?

Your comment I should have weighed with the known evidence of ..
**extreme** need for a smoke .. ?

Sooo .. would a decrease of receptors lead one to smoke an **extreme**
amount BECAUSE the receptors are not there .. ? .. IE: inhibition of
a7 nAChR

Which again would give credence to the lack of receptors DUE TO the
accumulated iron.

You smoke BECAUSE of a lack of receptors in order to simply function.

"In addition, inhibition of a7 nAChR at protein level was observed
in the cells exposed to high amounts of iron."

Who loves ya.
Tom

Jesus Was A Vegetarian!http://tinyurl.com/634q5a

Man Is A Herbivore!http://tinyurl.com/4rq595

DEAD PEOPLE WALKINGhttp://tinyurl.com/zk9fk- Hide quoted text -

- Show quoted text -[/quote]

ironjustice@aol.com · Guest

On Nov 30, 6:59 am, "ironjust...@aol.com" <ironjust...@aol.com> wrote:
"More than half of Parkinson>s patients smoke, as do half of
adolescents with severe Attention Deficit Hyperactivity Disorder
(ADHD)." <<

Lecithin in the treatment of mania: double-blind, placebo-controlled
trials
BM Cohen, JF Lipinski and RI Altesman

The authors report double-blind, placebo-controlled trials of pure
lecithin in the treatment of mania. As in preliminary trials, lecithin
appeared to be nontoxic and effective. Improvement with lecithin was
significantly greater than improvement with placebo in five of the six
patients studied. The concurrent use of anticholinergic agents did not
prevent the antimanic effect of lecithin. The authors discuss the
possible mechanism of action of lecithin, including cholinergic and
membrane-altering effects.
Am J Psychiatry 1982; 139:1162-1164
Copyright © 1982 by American Psychiatric Association

--------------------------------------------------------------------------------

-----------------

Who loves ya.
Tom

Jesus Was A Vegetarian!
http://tinyurl.com/2r2nkh

Man Is A Herbivore!
http://tinyurl.com/a3cc3

DEAD PEOPLE WALKING
http://tinyurl.com/zk9fk

[quote]
http://www.thelecithinstore.com/shop/custom.asp?recid=7

Brain biochemists are finding increasing evidence that a variety of
brain and bodily disorders may result from the inability of the brain
to produce or utilize sufficient acetylcholine.

The impact of these discoveries was summarized by one of the MIT
researchers when he recommended at a symposium on Dietary choline
Sources and Brain Function held at MIT in November 1977, “it comes as
a surprise that nature would allow our dietary habits to control such
a vital function as neurotransmitter synthesis.”
-----------------

Who loves ya.
Tom

Jesus Was A Vegetarian!http://tinyurl.com/2r2nkh

Man Is A Herbivore!http://tinyurl.com/a3cc3

DEAD PEOPLE WALKINGhttp://tinyurl.com/zk9fk

On Nov 29, 5:58 pm, ironjustice <teamtan...@hotmail.com> wrote:More
not less receptors should be associated with increased smoking.

It seems this smoking IS .. coincidentally common ALSO to .. ADHD and
Parkinsons' .

"More than half of Parkinson>s patients smoke, as do half of
adolescents with severe Attention Deficit Hyperactivity Disorder
(ADHD)."

Therefore associated and evidenced to be affected when one targets the
acetylcholine pathway / IE: nicotine and choline activate
receptors ...

IF the 61 year old woman with "brain iron accumulation" .. was ..
ACTUALLY .. simply Parkinsons' / misdiagnosis / common cause .. "

One can OBVIOUSLY **see** .. her cure JUST like in MS and marijuana /
IE: tremors and symptoms disappear .

No denying what you .. see.

Iron COULD 'possibly' .. be involved in diseases which may .. share ..
a common treatment and therefore possibly a common .. cause.

Parkinsons and / or brain iron accumulation is reversed by iron
removal and they walk and talk normally PROVING .. imho .. a cure can
be effected.

THEREFORE a cure should be and can be .. expected .. IN .. OTHER ..
diseases which may share a common .. cause .. IE: iron ..

Parkinsons .. ? .. or brain iron accumulation .. ? .. or
schizophrenia .. ? or ADHD .. ? .. or Alzheimers .. ?

http://www.cosmosmagazine.com/features/print/2349/nicotine-can-it-sav...

Nicotine: can it save your brain?
Issue 23 of Cosmos, October 2008
by Becky McCall

-----------------

Who loves ya.
Tom

Jesus Was A Vegetarian!http://tinyurl.com/2r2nkh

Man Is A Herbivore!http://tinyurl.com/a3cc3

DEAD PEOPLE WALKINGhttp://tinyurl.com/zk9fk

On Nov 27, 5:26 pm, m...@otr.com wrote:
"Substantiates 7 nicotinic acetylcholine receptor in schizophrenia"
Not according to what the abstract you posted said:
"Recent studies indicate an association of the neuronal 7 nicotinic
acetylcholine receptor (7 AChR) with several aspects of
schizophrenia,
and decreased levels of this receptor in postmortem brains of
schizophrenic patients have been reported. In view of that and in"

More not less receptors should be associated with increased smoking.

I should read your post more .. closely ..

The should part ..

As pointed out .. they smoke four times more so there has to be a
connection to the nicotine receptors .. ?

Your comment I should have weighed with the known evidence of ..
**extreme** need for a smoke .. ?

Sooo .. would a decrease of receptors lead one to smoke an **extreme**
amount BECAUSE the receptors are not there .. ? .. IE: inhibition of
a7 nAChR

Which again would give credence to the lack of receptors DUE TO the
accumulated iron.

You smoke BECAUSE of a lack of receptors in order to simply function.

"In addition, inhibition of a7 nAChR at protein level was observed
in the cells exposed to high amounts of iron."

Who loves ya.
Tom

Jesus Was A Vegetarian!http://tinyurl.com/634q5a

Man Is A Herbivore!http://tinyurl.com/4rq595

DEAD PEOPLE WALKINGhttp://tinyurl.com/zk9fk-Hide quoted text -

- Show quoted text -- Hide quoted text -

- Show quoted text -[/quote]

ironjustice@aol.com · Guest

On Nov 30, 7:14 pm, "ironjust...@aol.com" <ironjust...@aol.com> wrote:
[quote]On Nov 30, 6:59 am, "ironjust...@aol.com" <ironjust...@aol.com> wrote:
[/quote]
Electrophilic methyl groups present in the diet ameliorate
pathological
states induced by reductive and oxidative stress: a hypothesis
MikloÂs Ghyczy1* and MihaÂly Boros2
1RhoÃne-Poulenc Rorer Co., Cologne, Germany
2Institute of Surgical Research, University of Szeged, Hungary
(Received 10 April 2000 ± Revised 19 September 2000 ± Accepted 16
October 2000)
Reductive stress, characterised by an increased NADH:NAD+ ratio, may
be as
common and as important a consequence of redox imbalance as oxidative
stress. It
may also be an important predisposing cause of the generation of
reactive oxygen
species. Considerable experimental and indirect clinical evidence
suggests that
protection against reductive stress depends on biomolecules with
electrophilic methyl
groups (EMG) such as S-adenosylmethionine, betaine, carnitine and
phosphatidylcholine.
Pathological processes leading to reductive stress and their relief by
such
protective agents is reviewed and the proposed molecular mechanism is
outlined.
These and other EMG-containing biomolecules are part of the daily diet
and may
represent an important control system for redox balance.
Reductive and oxidative stress: Dietary electrophilic methyl groups:
NADH: Methane:
Functional food
British Journal of Nutrition (2001), 85, 409±414 DOI: 10.1079/
BJN2000274
q Nutrition Society 2001-----------------

Who loves ya.
Tom

Jesus Was A Vegetarian!
http://tinyurl.com/2r2nkh

Man Is A Herbivore!
http://tinyurl.com/a3cc3

DEAD PEOPLE WALKING
http://tinyurl.com/zk9fk

[quote] "More than half of Parkinson>s patients smoke, as do half of
adolescents with severe Attention Deficit Hyperactivity Disorder
(ADHD)."

Lecithin in the treatment of mania: double-blind, placebo-controlled
trials
BM Cohen, JF Lipinski and RI Altesman

The authors report double-blind, placebo-controlled trials of pure
lecithin in the treatment of mania. As in preliminary trials, lecithin
appeared to be nontoxic and effective. Improvement with lecithin was
significantly greater than improvement with placebo in five of the six
patients studied. The concurrent use of anticholinergic agents did not
prevent the antimanic effect of lecithin. The authors discuss the
possible mechanism of action of lecithin, including cholinergic and
membrane-altering effects.
Am J Psychiatry 1982; 139:1162-1164
Copyright © 1982 by American Psychiatric Association

--------------------------------------------------------------------------------

-----------------

Who loves ya.
Tom

Jesus Was A Vegetarian!http://tinyurl.com/2r2nkh

Man Is A Herbivore!http://tinyurl.com/a3cc3

DEAD PEOPLE WALKINGhttp://tinyurl.com/zk9fk

http://www.thelecithinstore.com/shop/custom.asp?recid=7

Brain biochemists are finding increasing evidence that a variety of
brain and bodily disorders may result from the inability of the brain
to produce or utilize sufficient acetylcholine.

The impact of these discoveries was summarized by one of the MIT
researchers when he recommended at a symposium on Dietary choline
Sources and Brain Function held at MIT in November 1977, “it comes as
a surprise that nature would allow our dietary habits to control such
a vital function as neurotransmitter synthesis.”
-----------------

Who loves ya.
Tom

Jesus Was A Vegetarian!http://tinyurl.com/2r2nkh

Man Is A Herbivore!http://tinyurl.com/a3cc3

DEAD PEOPLE WALKINGhttp://tinyurl.com/zk9fk

On Nov 29, 5:58 pm, ironjustice <teamtan...@hotmail.com> wrote:More
not less receptors should be associated with increased smoking.

It seems this smoking IS .. coincidentally common ALSO to .. ADHD and
Parkinsons' .

"More than half of Parkinson>s patients smoke, as do half of
adolescents with severe Attention Deficit Hyperactivity Disorder
(ADHD)."

Therefore associated and evidenced to be affected when one targets the
acetylcholine pathway / IE: nicotine and choline activate
receptors ...

IF the 61 year old woman with "brain iron accumulation" .. was ..
ACTUALLY .. simply Parkinsons' / misdiagnosis / common cause .. "

One can OBVIOUSLY **see** .. her cure JUST like in MS and marijuana /
IE: tremors and symptoms disappear .

No denying what you .. see.

Iron COULD 'possibly' .. be involved in diseases which may .. share ...
a common treatment and therefore possibly a common .. cause.

Parkinsons and / or brain iron accumulation is reversed by iron
removal and they walk and talk normally PROVING .. imho .. a cure can
be effected.

THEREFORE a cure should be and can be .. expected .. IN .. OTHER ..
diseases which may share a common .. cause .. IE: iron ..

Parkinsons .. ? .. or brain iron accumulation .. ? .. or
schizophrenia .. ? or ADHD .. ? .. or Alzheimers .. ?

http://www.cosmosmagazine.com/features/print/2349/nicotine-can-it-sav....

Nicotine: can it save your brain?
Issue 23 of Cosmos, October 2008
by Becky McCall

-----------------

Who loves ya.
Tom

Jesus Was A Vegetarian!http://tinyurl.com/2r2nkh

Man Is A Herbivore!http://tinyurl.com/a3cc3

DEAD PEOPLE WALKINGhttp://tinyurl.com/zk9fk

On Nov 27, 5:26 pm, m...@otr.com wrote:
"Substantiates 7 nicotinic acetylcholine receptor in schizophrenia"
Not according to what the abstract you posted said:
"Recent studies indicate an association of the neuronal 7 nicotinic
acetylcholine receptor (7 AChR) with several aspects of
schizophrenia,
and decreased levels of this receptor in postmortem brains of
schizophrenic patients have been reported. In view of that and in"

More not less receptors should be associated with increased smoking.

I should read your post more .. closely ..

The should part ..

As pointed out .. they smoke four times more so there has to be a
connection to the nicotine receptors .. ?

Your comment I should have weighed with the known evidence of ..
**extreme** need for a smoke .. ?

Sooo .. would a decrease of receptors lead one to smoke an **extreme**
amount BECAUSE the receptors are not there .. ? .. IE: inhibition of
a7 nAChR

Which again would give credence to the lack of receptors DUE TO the
accumulated iron.

You smoke BECAUSE of a lack of receptors in order to simply function.

"In addition, inhibition of a7 nAChR at protein level was observed
in the cells exposed to high amounts of iron."

Who loves ya.
Tom

Jesus Was A Vegetarian!http://tinyurl.com/634q5a

Man Is A Herbivore!http://tinyurl.com/4rq595

DEAD PEOPLE WALKINGhttp://tinyurl.com/zk9fk-Hidequoted text -

- Show quoted text -- Hide quoted text -

- Show quoted text -- Hide quoted text -

- Show quoted text -[/quote]

ironjustice@aol.com · Guest

On Dec 1, 10:15 am, Pramesh Rutaji <p297tongue6...@newsguy.com> wrote:
Out here, you>re treading in the public commons. <<

No loons .. allowed ..

That seems to be a rule you don>t agree with .. ?

Why don>t you agree a rule should exist to no posting if you are a
loon .. ?

It would seem to me to be one of the obvious reasons why a person
shouldn>t be allowed to post to a thread .. ?

If you act like a loon .. there are OTHER groups ..

AND threads ..

MY threads .. no loons .. allowed ..

THAT is enforced BY .. ? .. observation ..

If one observes one consistently acting like a loon .. one must make a
decision as to the .. contributive capacity capabilities .. OF .. such
a person .. and then .. take appropriate actions ..

On my threads I just don>t read their bullsht .. and get rid of
them ..

They have no reason to be on my threads ..

There are other threads ..

Do you understand .. urine drinking man .. ?

Do ya .. ?

I believe I am most accomodating .. don>t you mr .. urine drinking
man .. ?

-----------------

Who loves ya.
Tom

Jesus Was A Vegetarian!
http://tinyurl.com/2r2nkh

Man Is A Herbivore!
http://tinyurl.com/a3cc3

DEAD PEOPLE WALKING
http://tinyurl.com/zk9fk

ironjustice@aol.com · Guest

On Nov 30, 7:36 pm, "ironjust...@aol.com" <ironjust...@aol.com> wrote:
Electrophilic methyl groups present in the diet ameliorate
pathological states induced by reductive and oxidative stress: a
hypothesis <<

Nutrition Discussion Forum
Evidence in support of a concept of reductive stress;
It is generally believed that free radical damage to
biological systems is exerted by the oxidative action of
hydroxyl radicals.
Some researchers have, however, observed that many
degenerative diseases are associated with a hypoxic
state that results in an increased NADH:
NAD+ ratio, leading to a reductive cytosolic environment
(Ido et al. 1997).
Yet a number of natural and synthetic substances shown
to be beneficial in such pathological states have been
paradoxically termed `antioxidants' despite the fact that they
exist in oxidized form, e.g. lipoic acid.
It has been recently hypothesized (Ghyczy & Boros,
2001) that biomolecules with a positively charged N or S
atom and a bound methyl group, termed electrophilic
methyl group, react with NADH thus ameliorating
reductive stress.
In this letter, preliminary data are presented in support
of the contention of Ghyczy & Boros (2001) that reductive
rather than oxidative stress is of clinical importance.
An increasing interest in oxidative stress in diabetes
mellitus (Lipinski, 2001) led to a search for a diagnostically
useful method to measure the antioxidative potential of
plasma.
To generate hydroxyl radicals, ascorbic acid and cupric
chloride were used.
Unexpectedly, when these two substances were added in
equimolar proportion to normal human plasma, a precipitate
was formed containing mostly aggregated fibrinogen.
To further investigate this phenomenon, a solution of purified
fibrinogen was used and was found to form insoluble precipitate
with ascorbic acid± cupric chloride mixture, however, at a
concentration 10- fold lower than needed in plasma.
The factor responsible for the inhibition of fibrinogen aggregation
in plasma was then identified as human serum albumin.
Although human serum albumin has been suggested to be an
antioxidant due to the presence of one -SH group, all other
thirty-four cysteines exist in the oxidized form as disulfides.
Hence, this protein is rather unlikely to act as an effective
antioxidant agent.
To elucidate a mechanism by which human serum
albumin inhibits hydroxyl radical-induced fibrinogen
aggregation, a spectrophotometric experiment was done
in which ascorbic acid±cupric chloride was added to a
solution of NAD and optical density recorded at 340 nm.
A rapid increase in optical density proved that the hydroxyl
radicals generated in this system have a reducing potential
that was inhibited by the addition of human serum albumin.
Molar proportions of the reagents used in the present
experiment revealed that one molecule of human serum
albumin neutralized fifteen hydroxyl radicals, indicating
that about one-half of disulfide bonds in this protein
underwent reduction.
It was previously observed that hydroxyl radical modification
of human serum albumin led to the formation of higher molecular
mass complexes (Davies & Delsignore, 1987), and that limited
reduction with dithiothreitol caused its aggregation and
coprecipitation with other plasma proteins (Lipinski & Egyud, 1992).
Consequently, human serum albumin can be considered as
a sacrificial antireductive protein which when modified by
hydroxyl radicals gives a signal to proteolytic degradation
and elimination from the circulation.
In conclusion, in view of the findings reported by
Ghyczy & Boros (2001) and the results presented in this
present letter, the concept of oxidative stress and
antioxidants needs be revised.
Not only electrophilic methyl group biomolecules, but many
other substances containing reducible groups may fall into
a category ofantireductants rather than antioxidants.
For example, highly unsaturated eicosapentanoic acid also
inhibited hydroxylradical-induced fibrinogen aggregation.
In addition, reductive addition of hydroxyl groups to double
bonds of pyrimidines, pyrazines, and other aromatic rings
may explain beneficial action of water soluble vitamins in free
radical-induced stress (Moorthy & Hayon, 1976).
Finally, similar reductive mechanisms may be involved in the
formation of hydroxy derivatives of nucleic acids, e.g. 8-
hydroxy-20-deoxyguanosine, generally albeit speculatively,
believed to be a result of oxidative stress
(Park & Floyd,
1994).
B. Lipinski
Department of Genetics and Epidemiology
Joslin Diabetes Center
Harvard Medical School
One Joslin Place
Boston
MA 02215
USA
DOI: 10.1079/BJN2001435
References
Davies KJA & Delsignore ME (1987) Protein damage and
degradation by oxygen radicals. Journal of Biological Chemistry
262, 9908±9913.
Ghyczy M & Boros M (2001) Electrophilic methyl groups present
in the diet ameliorate pathological states induced by reductive
and oxidative stress; a hypothesis. British Journal of Nutrition
85, 409±414.
Ido Y, Kilo C & Williamson JR (1997) Cytosolic NADH/NAD+,
free radicals, and vascular dysfunction in early diabetes
mellitus. Diabetologia 40, S115±S117.
------------------------

Evidence in support of a concept of reductive stress ±
Reply by Ghyczy & Boros
We are writing to agree with Dr B Lipinski>s perceptive
comments (Lipinski, 2002) and to suggest that they are of
wide relevance.
In particular, the importance of reductive stress should be
more widely recognized.
The prevailing view is extraordinarily blinkered.
In PubMed (2001) there are 8957 citations to oxidative stress
and none to reductive stress.
This reflects not reality, but the mistaken but widely
held ideas that oxidative stress is common, that it is the
main source of biological free radicals, and that it is (or
should be) susceptible to correction with antioxidants.
In fact, extensive clinical and experimental work over the past
30 years has failed to reveal a single abnormal clinical state
which could be confidently ascribed to oxidative stress or,
more importantly, which has convincingly benefited from
antioxidants.
One reason is probably the initial difficulty of grasping the
concept of reductive stress: another the relative paucity of
experimental methods for demonstrating and measuring it.
Reductive stress can be defined as an abnormally
increased electron pressure or `reducing power' and it can
occur either as a result of pathological processes leading to
an excess of high-energy reducing electrons (as in NADH),
a failure of mechanisms available for dealing with this rise
in electron pressure, or, occasionally perhaps, a combination
of both.
It is probably not only more common thanoxidative stress but
it is also the main source of reactive oxygen species.
We have suggested in our previous paper
(Ghyczy & Boros, 2001): (1) that biomolecules with
electrophilic methyl groups are potential electron acceptors
and probably the main natural defences against reductive
stress.
Biomolecules which posses such electrophilic methyl groups
are S-adenosylmethionine, betaine, carnitine,
choline, glycerylphosphocholine, phosphatidylcholine,
which have a trimethylnitrogen moiety.
Of course there may be others.
The manner in which they exercise this vital function is now
under investigation.
However, the benefits of a such nutrients are being
recognized.
Most importantly, in 1998, the National Academy of Sciences,
USA, determined that choline is an essential vitamin
(United States Food and Drug Administration,
2001), and the US Food and Drug Administration
has recently agreed to allow new food labels to indicate
choline content with phrases such as: `a good source of
choline' (United States Food and Drug Administration,
2001)
Our preliminary results suggest that electrophilic
methyl group-containing biomolecules, such as choline,
may fulfil the criteria for a new class of functional food for
the control of intracellular redox balance.

MikloÂs Ghyczy
RhoÃne-Poulenc-Rorer Co.
Cologne
Germany
MihaÂly Boros
Institute of Surgical Research
University of Szeged
Hungary
DOI: 10.1079/BJN2001488
References
Ghyczy M & Boros M (2001) Electrophilic methyl groups
present in the diet ameliorate pathological states induced by
reductive and oxidative stress: a hypothesis. British Journal of
Nutrition 85, 409±414.
Lipinski B (2002) Evidence in support of a concept of reductive
stress. British Journal of Nutrition 87, 93±94.
PubMed (2001) http://www.ncbi.nlm.nih.gov/PubMed/
United States Food and Drug Administration (2001) Nutrient
Content Claims Notification for Choline Containing
Foods.-----------------

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Pramesh Rutaji · Guest

ironjustice wrote:
[quote]On Nov 28, 6:50 am, anonym...@nowhere.you.know wrote: snip

You were told to stay off my threads dioxin boy ..

[/quote]
I don>t have you filtered but this persisting bullshit is going to be
sufficient. Just because you don>t want any one to challenge your
extreme dogma. If you want your OWN thread, contract with and ISP to
provide your own message boards.

Out here, you>re treading in the public commons.

--

Pramesh Rutaji

p297tongue6221@newsguy.com - remove tongue to reply

steave jackson · Guest

On Sun, 30 Nov 2008 19:14:53 -0800 (PST), in alt.support.schizophrenia
"ironjustice@aol.com" <ironjustice@aol.com> wrote:

[quote]On Nov 30, 6:59Â am, "ironjust...@aol.com" <ironjust...@aol.com> wrote:
"More than half of Parkinson>s patients smoke, as do half of
adolescents with severe Attention Deficit Hyperactivity Disorder
(ADHD)."

Lecithin in the treatment of mania: double-blind, placebo-controlled
trials
BM Cohen, JF Lipinski and RI Altesman

The authors report double-blind, placebo-controlled trials of pure
lecithin in the treatment of mania. As in preliminary trials, lecithin
appeared to be nontoxic and effective. Improvement with lecithin was
significantly greater than improvement with placebo in five of the six
patients studied. The concurrent use of anticholinergic agents did not
prevent the antimanic effect of lecithin. The authors discuss the
possible mechanism of action of lecithin, including cholinergic and
membrane-altering effects.
Am J Psychiatry 1982; 139:1162-1164
Copyright Â© 1982 by American Psychiatric Association

--------------------------------------------------------------------------------
[/quote]
ok so how do I get my hands of some of that?

steave jackson · Guest

don>t post this insulting shit to alt.support.schizophrenia!

On Mon, 1 Dec 2008 13:51:33 -0800 (PST), in alt.support.schizophrenia
"ironjustice@aol.com" <ironjustice@aol.com> wrote:

[quote]On Dec 1, 10:15Â am, Pramesh Rutaji <p297tongue6...@newsguy.com> wrote:
Out here, you>re treading in the public commons.

No loons .. allowed ..

That seems to be a rule you don>t agree with .. ?

Why don>t you agree a rule should exist to no posting if you are a
loon .. ?

It would seem to me to be one of the obvious reasons why a person
shouldn>t be allowed to post to a thread .. ?

If you act like a loon .. there are OTHER groups ..

AND threads ..

MY threads .. no loons .. allowed ..

THAT is enforced BY .. ? .. observation ..

If one observes one consistently acting like a loon .. one must make a
decision as to the .. contributive capacity capabilities .. OF .. such
a person .. and then .. take appropriate actions ..

On my threads I just don>t read their bullsht .. and get rid of
them ..

They have no reason to be on my threads ..

There are other threads ..

Do you understand .. urine drinking man .. ?

Do ya .. ?

I believe I am most accomodating .. don>t you mr .. urine drinking
man .. ?

-----------------

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DEAD PEOPLE WALKING
http://tinyurl.com/zk9fk
[/quote]

ironjustice@aol.com · Guest

On Dec 1, 2:13 pm, "ironjust...@aol.com" <ironjust...@aol.com> wrote:
Electrophilic methyl groups present in the diet ameliorate
pathological states induced by reductive and oxidative stress: a
hypothesis <<

Autism: Amino acid DMG helps in 42 per cent of cases

Published on Saturday, March 29, 2008
by Healthy News Service

Children with autism can be helped by taking the amino acid DMG
(dimethylglycine), a major new study has confirmed. The benefits of
the supplement – especially for people with mental problems - have
been known for years, but the evidence has either been anecdotal or
the studies have been too small to interest other researchers.
The new study, organised by the Autism Research Institute, recruited
5,367 autistic children – and 42 per cent of them reported major
improvements in their condition after taking the supplement.

According to teachers and parents, the children who responded
positively to DMG had improved verbal communication, better social
interaction, better eye contact, improved affection, a reduction in
seizures and improved sleep patterns.

One of DMG’s long-time advocates, Dr. Roger V Kendall, says that it
helps the neurological system and can also modulate the immune system.
It acts as a methyl donor, and methylation activates neural pathways
that affect behaviour.

He recommends one to four 125 mg DMG tablets per day for an autistic
child, and it should be taken with 800 micrograms of folic acid. DMG
can also be found naturally in liver, and in beans, seeds and grains.

(Source: Townsend Letter, 2008; 297: 34).

Who loves ya.
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Man Is A Herbivore!
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DEAD PEOPLE WALKING
http://tinyurl.com/zk9fk

ironjustice@aol.com · Guest

On Dec 1, 8:58 pm, "ironjust...@aol.com" <ironjust...@aol.com>
wrote:the amino acid DMG (dimethylglycine), <<

--------------------------------------------------------------------------------
Autism Research Review International, 1990, Vol. 4, No. 2, page 3

Dimethylglycine (DMG), a Nontoxic Metabolite, and Autism
DMG is a rather sweet-tasting substance that was described in a recent
article in the Journal of Laboratory and Clinical Medicine (1990,
481-86) as a “natural, simple compound with no known undesirable side
effects.” The article did not pertain to the use of DMG in autism, but
instead described an experiment in which DMG was used to try to
enhance the function of the immune system of laboratory rabbits. It
worked—the immune systems of the animals given DMG showed 300% to
1,000% better response to infection than the controls.

DMG is readily available in many health food stores. It is legally
classified as a food. It does not require a prescription. It is
manufactured by several companies, and comes in various forms, most
commonly in tiny foil-wrapped tablets about 1/3 the size of an
aspirin.

The taste is pleasant and children chew the tablets readily. At about
25 cents per tablet, the cost is minimal, since only one to eight
tablets a day are usually taken (eight for adults).

“So far so good,” you may be saying, “but what does this have to do
with autism?”

In 1965, two Russian investigators, M. G. Blumena and T. L. Belyakova,
published a report showing considerable improvement in the speech of
12 of a group of 15 mentally handicapped children who had not been
able to use speech to communicate. The children had been treated with
a substance variously known as calcium pangamate, or pangamic acid, or
“vitamin B15.” In addition to enriched vocabulary, the children began
to use simple sentences, their general mental state improved, and
there was better concentration and interest in toys and games.
Subsequent research has shown the essential factor in calcium
pangamate to be DMG.

Soon afterward psychiatrist Allan Cott visited Moscow and brought back
a small supply of pangamic acid, which he tried on a number of
children in his practice, some of whom were autistic. Many of Cott’s
patients responded in the same way the Russian children had. One
mother wrote, “It’s the most exciting thing I’ve ever experienced. He
was repeating words and he answers questions now….”

At about this time pangamic acid, or B15, entered the U.S. market.
Chaos ensued. Every manufacturer touted his product as “the original
Russian formula.” There were at least four different formulas on the
market, partly, it is believed, as a result of deliberate deception
and obfuscation on the part of the Russians. DMG, in small amounts,
was a component of some of the formulas. The FDA stepped in and
lengthy legal battles ensued. One outcome is that the term B15 was
outlawed. (Although DMG resembles the B vitamins in many ways—it is
found in the same foods, for example—there are no known overt symptoms
characteristic of a DMG deficiency.)

The significant outcome of the legal battles is that the sale of DMG
is now permitted, as long as it is not referred to as a vitamin, and
as long as it is sold as a food and not a drug.

I have been following the pangamic acid-DMG situation for almost 25
years. I have mentioned it in some of my lectures, and told parents
and professionals about it in conversations and correspondence. Always
I would ask, “if you try it, please let me know what results you see,
even if no improvement is found.”

I am now so firmly convinced that DMG is helpful to a substantial
proportion of autistic children and adults that I have decided to “go
public” in the Autism Research Review International —to tell people
about it freely and openly, so they may try it if they wish.

Some who hear of this boldness may be aghast: “Where are the double
blind placebo-controlled scientific studies showing it to be effective
in autism?” they will ask. My reply is simple. “There aren’t any, and
none are needed.” There are, of course, numerous double blind non-
autism studies of DMG in the scientific and medical literature, using
not only humans, but many kinds of laboratory animals, often given
very large amounts of DMG. As noted earlier, no adverse side effects
have been found with even massive intakes of DMG. (I say “intakes”
rather than “dosages” because “dosage” implies that DMG is a drug,
which it is not.)

Since no company has the exclusive right to make DMG, competition
keeps the price—and profits—down. Thus there is almost no chance that
anyone will sponsor a $200,000 double blind study of DMG on autistic
children. A parent can buy 30 tablets for about $8.00. That is a
sufficient supply, even for an adult given five or more tablets a day,
to determine, in most cases, if it will be helpful. If it is felt to
be helpful, fine. If not, you have wasted $8.00 (except for the boost
given to the immune system).

To help the parents receive unbiased input, I usually tell them to
refrain from mentioning to teachers, grandparents and others in the
child’s environment that DMG is being tried. I have numerous letters
in my files saying, “Johnny’s speech therapist says he has made more
progress in the last two weeks than in the last six months. As you
suggested, we had told no one at his school that we were trying DMG.”

I am 100% in favor of double blind studies on drugs with considerable
potential for harm, such as fenfluramine, Haldol, or the like.
However, it doesn’t make sense to insist on such refinements before
trying a perfectly safe substance such as DMG, apple pie, or chicken
soup.

If DMG is going to work, its effects will usually be seen within a
week or so, though it should be tried for a few weeks or a month
before giving up. In some cases dramatic results have been seen within
24 hours: A Los Angeles mother was driving on the freeway, three-year-
old Kathy in the back seat, five-year-old mute autistic son Sammy in
the front. DMG had been started the day before. Kathy began to cry.
Sammy turned and spoke his first words: “Don’t cry, Kathy.” The
mother, stunned, almost crashed the car.

A similar case: A Texas mother secured her six-year-old mute autistic
daughter in the front seat, then, before driving off, turned to tell
her husband, “I’ll drop Mary at the babysitter’s house first.” Mary,
on DMG for two days, startled her parents with her first words: “No!
No babysitter!”

Although speech is the most notable positive change in those children
helped by DMG, behavioral improvement is also often reported. One
father gave his son one DMG tablet per day without mentioning it to
the school. He later requested a copy of the school’s detailed record
of his son’s day-by-day behavioral transgressions. The correlation
between outburst-free days and the use of DMG was unmistakable.

An article in the New England Journal of Medicine (October 1982)
reported that a 22-year-old mentally retarded man who had 16 to 18
seizures per week on standard anticonvulsants, experienced only three
seizures per week while on DMG. Two attempts to remove the DMG
dramatically increased seizure frequency.

Last year I sent information on DMG to Lee Dae Kun, Director of the
Pusan (Korea) Research Center on Child Problems. He tried the DMG on
39 autistic children, ages three to seven, for three months, with the
following (summarized) results:

Benefits seen: Yes: 31 (80%)
No: 8 (20%)
(Improved speech, eating, excretion, willingness, etc.)
8 children had difficulty sleeping for weeks 1 and 2.
6 children became more active for weeks 1 and 2.

Lee Dae Kun wrote that the parents, usually skeptical, saw the
improvements clearly. He concluded that DMG is very beneficial for
children with autism, even if it is not a cure.

Information about the use of DMG with older persons is also
encouraging. One mother of a 26-year-old who squeezed things (people,
TV sets, etc.) very hard when frustrated, tried DMG, quite
skeptically, to see if it would stimulate his very sparse speech. It
didn’t, but brought remarkable improvement in his frustration
tolerance. “Even my husband, who was even more skeptical than me, now
is a believer,” she wrote. DMG certainly doesn’t always help, and it
certainly is not a cure, but it is certainly worth trying, in my
humble opinion.

If you try it, let me hear from you.

© 2007 Autism Research Institute | Notices | Webcasts

Who loves ya.
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Jesus Was A Vegetarian!
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http://tinyurl.com/a3cc3

DEAD PEOPLE WALKING
http://tinyurl.com/zk9fk

[quote]On Dec 1, 2:13 pm, "ironjust...@aol.com" <ironjust...@aol.com> wrote:
Electrophilic methyl groups present in the diet ameliorate
pathological states induced by reductive and oxidative stress: a
hypothesis

Autism: Amino acid DMG helps in 42 per cent of cases

Published on Saturday, March 29, 2008
by Healthy News Service

Children with autism can be helped by taking the amino acid DMG
(dimethylglycine), a major new study has confirmed. The benefits of
the supplement – especially for people with mental problems - have
been known for years, but the evidence has either been anecdotal or
the studies have been too small to interest other researchers.
The new study, organised by the Autism Research Institute, recruited
5,367 autistic children – and 42 per cent of them reported major
improvements in their condition after taking the supplement.

According to teachers and parents, the children who responded
positively to DMG had improved verbal communication, better social
interaction, better eye contact, improved affection, a reduction in
seizures and improved sleep patterns.

One of DMG’s long-time advocates, Dr. Roger V Kendall, says that it
helps the neurological system and can also modulate the immune system.
It acts as a methyl donor, and methylation activates neural pathways
that affect behaviour.

He recommends one to four 125 mg DMG tablets per day for an autistic
child, and it should be taken with 800 micrograms of folic acid. DMG
can also be found naturally in liver, and in beans, seeds and grains.

(Source: Townsend Letter, 2008; 297: 34).

Who loves ya.
Tom

Jesus Was A Vegetarian!http://tinyurl.com/2r2nkh

Man Is A Herbivore!http://tinyurl.com/a3cc3

DEAD PEOPLE WALKINGhttp://tinyurl.com/zk9fk[/quote]

ironjustice@aol.com · Guest

On Dec 1, 8:58 pm, "ironjust...@aol.com" <ironjust...@aol.com> wrote:
Children with autism can be helped by taking the amino acid DMG
(dimethylglycine), <<

It seems choline raises its' .. head ..

http://www.vitalhealthzone.com/nutrition/other-nutrients/dimethyl_glycine.html

"Why dimethyl glycine is good for you

Dimethyl Glycine is produced in cells as a mid-way in the metabolism
of choline to glycine. It acts as a building block for the
biosynthesis of many important substances such as methionine, choline,
several important hormones, neurotransmitters, and DNA."

Who loves ya.
Tom

Jesus Was A Vegetarian!
http://tinyurl.com/2r2nkh

Man Is A Herbivore!
http://tinyurl.com/a3cc3

DEAD PEOPLE WALKING
http://tinyurl.com/zk9fk

[quote]a major new study has confirmed. The benefits of
the supplement – especially for people with mental problems - have
been known for years, but the evidence has either been anecdotal or
the studies have been too small to interest other researchers.
The new study, organised by the Autism Research Institute, recruited
5,367 autistic children – and 42 per cent of them reported major
improvements in their condition after taking the supplement.

According to teachers and parents, the children who responded
positively to DMG had improved verbal communication, better social
interaction, better eye contact, improved affection, a reduction in
seizures and improved sleep patterns.

One of DMG’s long-time advocates, Dr. Roger V Kendall, says that it
helps the neurological system and can also modulate the immune system.
It acts as a methyl donor, and methylation activates neural pathways
that affect behaviour.

He recommends one to four 125 mg DMG tablets per day for an autistic
child, and it should be taken with 800 micrograms of folic acid. DMG
can also be found naturally in liver, and in beans, seeds and grains.

(Source: Townsend Letter, 2008; 297: 34).

Who loves ya.
Tom

Jesus Was A Vegetarian!http://tinyurl.com/2r2nkh

Man Is A Herbivore!http://tinyurl.com/a3cc3

DEAD PEOPLE WALKINGhttp://tinyurl.com/zk9fk[/quote]

ironjustice@aol.com · Guest

On Nov 30, 7:14 pm, "ironjust...@aol.com" <ironjust...@aol.com> wrote:
"More than half of Parkinson>s patients smoke, as do half of
adolescents with severe Attention Deficit Hyperactivity Disorder
(ADHD)." <<

Iron in the brains of Parkinson>s disease sufferers
http://www.srs.dl.ac.uk/Annual_Reports/AnRep94_95/Ch2/Iron.html
--------------------------------------------------------------------------------
Studying the mechanism of iron storage in the brain will help to
understand the disease process.
--------------------------------------------------------------------------------

The accumulation of metals in brain tissue has frequently been
associated with neurological disorders - Alzheimer>s disease is linked
to aluminium, Wilson>s disease to copper and Hallervorden-Spatz
disease to iron. It has been known for some time that sufferers of
Parkinson>s disease also have abnormally high levels of iron in the
brain. Paul Griffiths of Newcastle General Hospital Department of
Neuroradiology, in collaboration with Barry Dobson and Gareth Jones at
Daresbury Laboratory, have used the SRS to investigate how iron is
stored in the brain in both healthy and Parkinson>s diseased tissue.
Now, for the first time, definite conclusions have been drawn about
the method of iron storage.

Iron is stored throughout the brain, accumulating rapidly during
adolescence and early adulthood. The areas containing most iron are at
the base of the brain - the substantia nigra and the globus pallidus -
and these areas are also thought to be affected most in degenerative
diseases. The presence of iron is perfectly normal; a healthy adult
brain might contain 50 mg of iron per gram of tissue. However, in
Parkinson>s sufferers, this figure can rise to 250 mg per gram of
tissue. Examining tissue from healthy brains and from Parkinson>s
sufferers, shows the similarities and differences in the iron
deposits.

A common method of iron storage in the body is via a protein called
ferritin. Ferritin is a large protein, like a molecular football, that
can hold 30 or 40 iron atoms inside. The iron is inert when it is
stored inside the ferritin. A technique called X-ray Absorption Fine
Structure (or XAFS) Spectroscopy has been used to study the brain
tissue (figure 1.35). XAFS 'homes in' on particular atoms in a sample,
and then identifies the position and type of their nearest neighbours.
Using this technique the environment of the iron atoms in the brain
can be studied in detail, clearly showing the signature of ferritin in
both healthy and diseased tissue. The brain iron can now be positively
identified as ferritin.

In addition to characterising the iron deposits with XAFS, the core
centres can be imaged using cryo electron microscopy.

Ferritin cores in the brain tissue show up clearly as dark spots on
the image (figure 1.36). The microscopy not only shows the location of
the cores, but also indicates their density. Here a clear difference
is seen between normal and Parkinson>s tissue, with the ferritin
'footballs' in diseased brains being more heavily loaded with iron.

The role of iron in Parkinson>s disease is not yet fully understood.
However, these experiments have given the first conclusive results of
the method of iron storage in the brain. This knowledge may improve
our understanding of the degenerative processes involved in
Parkinson>s disease.

For more information contact: Dr B.R. Dobson
Tel: 01925 603323 e-mail: b.r.dobson@dl.ac.uk
----------------------
Parkinson>s Disease Linked To High Iron Intake
ScienceDaily (June 10, 2003) — ST. PAUL, MN – People with high levels
of iron in their diet are more likely to develop Parkinson>s disease,
according to a study in the June 10 issue of Neurology, the scientific
journal of the American Academy of Neurology. People with both high
levels of iron and manganese were nearly two times more likely to
develop the disease than those with the lowest levels of the minerals
in their diets. The study compared 250 people who were newly diagnosed
with Parkinson>s to 388 people without the disease. Interviews were
conducted to determine how often participants ate certain foods during
their adult life.

--------------------------------------------------------------------------------
See also:
Health & Medicine
Parkinson>s Research
Dietary Supplement
Alzheimer>s Research
Mind & Brain
Parkinson>s
Alzheimer>s
Disorders and Syndromes
Reference
Dietary mineral
Micronutrient
Essential nutrient
Dementia with Lewy bodies
Those who had the highest level of iron in their diets – in the top 25
percent – were 1.7 times more likely to be Parkinson>s patients than
those in the lowest 25 percent of iron intake. Those whose level of
both iron and manganese was higher than average were 1.9 times more
likely to be Parkinson>s patients than those with lower than average
intake of the minerals.

Iron and manganese contribute to oxidative stress, a situation where
cells release toxic substances called free radicals as part of normal
energy consumption and metabolism.

"Oxidative stress may cause degeneration of brain cells that produce
dopamine – the same cells that are affected by Parkinson>s disease,"
said study author Harvey Checkoway, PhD, of the University of
Washington in Seattle.

People who had higher than average dietary iron intake and who also
took, on average, one or more multivitamins or iron supplements per
day were 2.1 times more likely to be Parkinson>s patients than those
who had lower than average dietary iron intake and who took fewer than
one multivitamin or iron supplement per day.

Those who had higher than average dietary manganese intake and also
took an average of one or more multivitamins per day were 1.9 times
more likely to be Parkinson>s patients than those who had lower than
average dietary manganese intake and who took less than one
multivitamin per day.

Additional studies are necessary to confirm these results, Checkoway
said.

Foods rich in both iron and manganese include spinach, legumes, nuts
and whole grains. Iron is also abundant in red meat and poultry.

Checkoway said that the benefits of eating foods rich in iron and
manganese and in taking multivitamins outweigh the risks of developing
Parkinson>s disease.

"Our findings may improve understanding of how Parkinson>s disease
develops," he said. "But, there are most likely numerous
environmental, lifestyle and genetic factors that determine who will
develop the disease. It>s too early to make any recommendations about
potential dietary changes."

The research was supported by grants from the National Institute of
Environmental Health Sciences.

The American Academy of Neurology, an association of more than 18,000
neurologists and neuroscience professionals, is dedicated to improving
patient care through education and research. A neurologist is a doctor
with specialized training in diagnosing, treating and managing
disorders of the brain and nervous system such as stroke, Alzheimer>s
disease, epilepsy, Parkinson>s disease, autism and multiple sclerosis.

--------------------------------------------------------------------------------

Adapted from materials provided by American Academy Of Neurology.
Email or share this story:
Need to cite this story in your essay, paper, or report? Use one of
the following formats:
APA

MLA American Academy Of Neurology (2003, June 10). Parkinson>s
Disease Linked To High Iron Intake. ScienceDaily. Retrieved December
2, 2008, from http://www.sciencedaily.com /releases/
2003/06/030610081311.htm
-----------------

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http://tinyurl.com/2r2nkh

Man Is A Herbivore!
http://tinyurl.com/a3cc3

DEAD PEOPLE WALKING
http://tinyurl.com/zk9fk

[quote]
Lecithin in the treatment of mania: double-blind, placebo-controlled
trials
BM Cohen, JF Lipinski and RI Altesman

The authors report double-blind, placebo-controlled trials of pure
lecithin in the treatment of mania. As in preliminary trials, lecithin
appeared to be nontoxic and effective. Improvement with lecithin was
significantly greater than improvement with placebo in five of the six
patients studied. The concurrent use of anticholinergic agents did not
prevent the antimanic effect of lecithin. The authors discuss the
possible mechanism of action of lecithin, including cholinergic and
membrane-altering effects.
Am J Psychiatry 1982; 139:1162-1164
Copyright © 1982 by American Psychiatric Association

--------------------------------------------------------------------------------

-----------------

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http://www.thelecithinstore.com/shop/custom.asp?recid=7

Brain biochemists are finding increasing evidence that a variety of
brain and bodily disorders may result from the inability of the brain
to produce or utilize sufficient acetylcholine.

The impact of these discoveries was summarized by one of the MIT
researchers when he recommended at a symposium on Dietary choline
Sources and Brain Function held at MIT in November 1977, “it comes as
a surprise that nature would allow our dietary habits to control such
a vital function as neurotransmitter synthesis.”
-----------------

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Tom

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Man Is A Herbivore!http://tinyurl.com/a3cc3

DEAD PEOPLE WALKINGhttp://tinyurl.com/zk9fk

On Nov 29, 5:58 pm, ironjustice <teamtan...@hotmail.com> wrote:More
not less receptors should be associated with increased smoking.

It seems this smoking IS .. coincidentally common ALSO to .. ADHD and
Parkinsons' .

"More than half of Parkinson>s patients smoke, as do half of
adolescents with severe Attention Deficit Hyperactivity Disorder
(ADHD)."

Therefore associated and evidenced to be affected when one targets the
acetylcholine pathway / IE: nicotine and choline activate
receptors ...

IF the61yearoldwomanwith "brain iron accumulation" .. was ..
ACTUALLY .. simply Parkinsons' / misdiagnosis / common cause .. "

One can OBVIOUSLY **see** .. her cure JUST like in MS and marijuana /
IE: tremors and symptoms disappear .

No denying what you .. see.

Iron COULD 'possibly' .. be involved in diseases which may .. share ...
a common treatment and therefore possibly a common .. cause.

Parkinsons and / or brain iron accumulation is reversed by iron
removal and they walk and talk normally PROVING .. imho .. a cure can
be effected.

THEREFORE a cure should be and can be .. expected .. IN .. OTHER ..
diseases which may share a common .. cause .. IE: iron ..

Parkinsons .. ? .. or brain iron accumulation .. ? .. or
schizophrenia .. ? or ADHD .. ? .. or Alzheimers .. ?

http://www.cosmosmagazine.com/features/print/2349/nicotine-can-it-sav....

Nicotine: can it save your brain?
Issue 23 of Cosmos, October 2008
by Becky McCall

-----------------

Who loves ya.
Tom

Jesus Was A Vegetarian!http://tinyurl.com/2r2nkh

Man Is A Herbivore!http://tinyurl.com/a3cc3

DEAD PEOPLE WALKINGhttp://tinyurl.com/zk9fk

On Nov 27, 5:26 pm, m...@otr.com wrote:
"Substantiates 7 nicotinic acetylcholine receptor in schizophrenia"
Not according to what the abstract you posted said:
"Recent studies indicate an association of the neuronal 7 nicotinic
acetylcholine receptor (7 AChR) with several aspects of
schizophrenia,
and decreased levels of this receptor in postmortem brains of
schizophrenic patients have been reported. In view of that and in"

More not less receptors should be associated with increased smoking.

I should read your post more .. closely ..

The should part ..

As pointed out .. they smoke four times more so there has to be a
connection to the nicotine receptors .. ?

Your comment I should have weighed with the known evidence of ..
**extreme** need for a smoke .. ?

Sooo .. would a decrease of receptors lead one to smoke an **extreme**
amount BECAUSE the receptors are not there .. ? .. IE: inhibition of
a7 nAChR

Which again would give credence to the lack of receptors DUE TO the
accumulated iron.

You smoke BECAUSE of a lack of receptors in order to simply function.

"In addition, inhibition of a7 nAChR at protein level was observed
in the cells exposed to high amounts of iron."

Who loves ya.
Tom

Jesus Was A Vegetarian!http://tinyurl.com/634q5a

Man Is A Herbivore!http://tinyurl.com/4rq595

DEAD PEOPLE WALKINGhttp://tinyurl.com/zk9fk-Hidequoted text -

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